GNAQ体细胞突变导致SWS的机制及MEK和PI3K抑制剂联合作用机制研究
关注'%3e%3cg%3e%3cpath%20d='M18,16L13,16L10,19L7,16L2,16L2,3L18,3L18,16ZM12.3787,14.5L10,16.878700000000002L7.62132,14.5L3.5,14.5L3.5,4.5L16.5,4.5L16.5,14.5L12.3787,14.5Z'%20fill-rule='evenodd'%20fill='%23FFFFFF'%20fill-opacity='1'/%3e%3c/g%3e%3cg%3e%3cg%3e%3cellipse%20cx='6'%20cy='9.5'%20rx='1'%20ry='1'%20fill='%23FFFFFF'%20fill-opacity='1'/%3e%3c/g%3e%3cg%3e%3cellipse%20cx='10'%20cy='9.5'%20rx='1'%20ry='1'%20fill='%23FFFFFF'%20fill-opacity='1'/%3e%3c/g%3e%3cg%3e%3cellipse%20cx='14'%20cy='9.5'%20rx='1'%20ry='1'%20fill='%23FFFFFF'%20fill-opacity='1'/%3e%3c/g%3e%3c/g%3e%3c/g%3e%3c/svg%3e)
联系合作 CGT（细胞与基因治疗）
成果单位： 首都医科大学附属北京儿童医院
合作方式： 合作开发
所处阶段： 概念
关键词： GNAQR183Q突变研究血管畸形疾病治疗细胞实验MEK抑制剂PI3K抑制剂信号通路磷酸化检测mRNA检测
总得分（满分100）
0
资本强度（满分0）
该成果得分：0
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